In my previous post (A Deeper Understanding of the Bat Spirit Hugrún), I shared that my own personality contains schizotypal and autistic features characteristic of the intimately intertwined spectrums of schizophrenia and autism, both of which are heritable conditions of consciousness. Both autism and schizophrenia are phenotypically expressed in males more than in females in a ratio of 4 males to every 1 female diagnosed.
In my own family, I have a maternal first cousin (male), a paternal great aunt and two paternal second cousins (both sons of the great aunt who had three children in total) - 3 males and 1 female with diagnoses schizophrenia. I have three adult children (2 males, 1 female) and eight grandchildren (6 males, 2 females), none of which manifest either autism or schizophrenia. I had one first trimester miscarriage (male). Then there is me, a female with high functioning autistic and schizotypal traits, but phenotypically expressing neither autism nor schizophrenia in fullness, yet in parallel claiming an atypical experience and memory of my own incarnating consciousness. How can this be rationally explained? I will take up the challenge.
The phenotypical expression of autism in males is known to be linked with Fragile X-Chromosome Syndrome (FXS). An X-chromosome is inherited from the mother by both males (XY) and females (XX). Females, who inherit two X-chromosomes, one from the father and one from the mother, transcriptionally inactivate one of the X-chromosomes.
The X-chromosome is scientifically linked with working memory. In an earlier post (X-Chromosome Mediated Genetic Sensory Memory), I hypothesized the presence of a 'strong' X-chromosome functionality to the atypical continuous memory of my own incarnation of conscious awareness (a continuous memory I came into incarnation carrying and continue to carry to this day). As the phenotypical expression of autism and schizophrenia express along a spectrum, so too may the relative functional 'fragility' and 'strength' of the active X-chromosome.
One genetic feature of FXS is the presence of a CGG codon reiteration in the genetic code on the X- chromosome. A codon is a series of three nucleotides which form a unit of genetic code. There are between 5 and 40 repeats of this codon in the FMR1 gene on the X- chromosome of most people ('normal' neurotypical persons). In FXS there are more than 200 of these codon repeats. The FMR1 protein (produced by the expression of the FMR1 gene) is required for the development of connections between neurons. The production of the FMR1 protein is greatly attenuated (reduced) when the number of these particular CGG codon repeats reaches 200.
While it is clear that neurotypical persons have between 5 and 40 CGG repeats on the FMR1 gene on the X-chromosome which is linked with both neural connectivity and working memory, and that persons with severe phenotypically expressed autism have over 200, the functional effect of phenotypic expression is scientifically unsettled in aneurotypical persons having between 40 and 200 of the CGG codon repeats. We can therefore logically ask, "what is the phenotypically expressed functional effect resulting in persons having between 40 to 200 of these CGG codon repeats?" Importantly, we can also ask, "is there an adaptive evolutionary benefit for preserving (and possibly selecting for) this redundancy of genetic code?"
Given the effect of the CGG codon repeat on neural connectivity, and therefore on neuroplasticity (the capacity of the neurons and neural networks in the brain to change their connections and behavior throughout an individual's life), I suggest that the adaptive evolutionary advantage of preserving the ability to produce this redundancy of genetic code may be the explosion of available information and its successful integration into biologically connected consciousness.
First, with the exponential explosion of both technology and information, the neuroplastic ability of a brain containing between 40 and 200 CGG codons may enable that brain to adaptively integrate more of this new information without disintegrating into intellectual overload as a consequence of being processed through relatively less plastic neural networks. I suggest that the range of between 40 to 200 of the CGG codon on the FMR1 gene is a 'goldilocks zone' for increasing the intellectual capacity of the human brain to deal with ever increasing knowledge of the cosmos.
Second, quantum physicists hypothesize the presence of parallel universes and the existence of different co-existent realities. Relatedly, spiritual and religious traditions across the world and throughout time have hypothesized the existence of different levels of the soul and consciousness. I suggest that the neuroplastic capability of the brain, particularly of a brain containing a goldilocks zone number of CGG codons in the FMR1 gene, enables the human biological system to incarnate and successfully integrate (into a functionally unified system) more sublime and complex levels of Consciousness, or of what I like to call, the Essential Soul Complex.
Taking all of this together, I suggest that the incarnation of more sublime levels of consciousness and a greater information processing capability of the brain is (at least in part) heritable and mediated by the active X-chromosome. More specifically, the CGG Codon Repeat in the FMR1 gene on the X-chromosome is significant to optimizing and increasing the brain's neuroplastic capability to integrate more sublime levels of consciousness, soul and cosmic information. Moreover, one clinical treatment for severe autism and schizophrenia may involve finding a way to augment the production of the FMR1 protein which is diminished in persons having over 200 of the CGG codon repeats on the FMR1 gene. Conversely, accessing more sublime levels of consciousness in neurotypical persons could involve a (natural or induced) mechanism which temporarily inhibits FMR1 protein binding in some amount.